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Volume 5, Issue 7 (Suppl)
J Infect Dis Ther, an open access journal
ISSN: 2332-0877
Infection Prevention 2017
December 14-15, 2017
December 14-15, 2017 | Rome, Italy
13
th
World Congress on
INFECTION PREVENTION AND CONTROL
Anti-biotics activity against isolates from patients surgical site infection at Gezira hospital for renal
diseases and surgery- Wad Medani, Sudan
Sitelbanat Yassin and Rasha Algam
University of Gezira, Sudan
W
ound infections contribute significantly to morbidity and mortality in surgically treated patients. Number of factors
contributes to wound infection; however, microorganisms are the major causes with bacteria being the most prevalent.
Determination of local bacterial sensitivity patterns to antibiotics is an important to provide a guide for an appropriate
management. The present study was designed to investigate the susceptibility of etiological pathogens isolated from the Gezira
Hospital for renal diseases and surgery, to different types of antimicrobial agents and the risk factors during the period between
August and December 2015. The study followed prospective case series for all patients admitted to the surgical wards during
this period. The demographic data, diagnostic criteria, and laboratory data were collected. Out of the 200 patients recruited in
the study, (18%) showed evidence of sepsis yield infections. The predominant isolate was
Staphylococcus aureus
which represent
(31%) of the total isolated Bacteria. Cloxacillin exhibited the highest activity (87%) against Staph aureus while Cefotaxime, Co-
trimoxazole and Cefalexin showed only (7%). In contrast to previous study reported that
Staph. aureus
was 100% susceptible
to Cefotaxime, this conflict may due to the production of β-lactamase or alternation of the target site with the decrease affinity
to the drug and the diabetic patients showed high incidence of wound infection compared with non-diabetic. In conclusion,
the rate of surgical site infection was relatively high, and the predominant pathogen
Staph. aureus
was highly susceptible to
Cloxacillin and resistant to Cefixim.
sitelbanatehaimir@gmail.comJ Infect Dis Ther 2017, 5:7(Suppl)
DOI: 10.4172/2332-0877-C1-036