global-physiotherapy-2016_posters-accepted-abstracts

16 / 16

Page 60
Global Physiotherapy 2016
November 17-18, 2016
Volume 6 Issue 6(Suppl) J Nov Physiother
ISSN: 2165-7025 JNP, an open access journal
conferenceseries .com
November 17-18, 2016 Atlanta, USA
Global Physiotherapy Congress
J Nov Physiother 2016, 6:6(Suppl)
Acomparative study on the influence of kinesio taping® and laser therapy on knee joint position sense,
pain intensity, and function in individuals with knee osteoarthritis
Vahid Mazloum
Azad University of Karaj, Iran
Introduction: Conservative rehabilitation methods are assumed as a fundamental part of treatment in patients with knee
osteoarthritis (OA). The objective was to investigate the influence of Kinesio Taping® (KT®) and low level laser therapy (LT) on
pain intensity, function, and knee joint position sense (JPS) in such patients.
Materials & Methods: Twenty-six male patients (Mean±SD of age: 48.5±4.6 years) with unilateral knee OA were randomly
divided in to two groups of KT® (N=13) and LT (N=13). Both groups followed their own specific therapeutic protocol in
addition to routine physiotherapy program for 10 sessions. The outcome measurements included pain intensity, function, and
knee JPS; which were evaluated using visual analogue scale, ‘Up and Go test’, and reproduction of target angle at baseline and
after completing the interventions; respectively.
Results: Bothmethods can significantly improve pain intensity, reduce the time to perform ‘Up andGo test’, and reduce the angle
reproduction error of 60º knee flexion (P<0.001). Mean difference for target angle reproduction error was more significant in
KT group compared to LT group (P<0.001); while no significant mean difference was found for other measurements (P>0.05).
Conclusion: KT® and low level laser can improve pain, knee JPS, and function in clients with knee OA; however there is better
effect of KT® on knee JPS.
Thermal medicine, the heat shock response and the modulation of inflammation: A therapeutic
come back in a remix
Wilton Remigio
Misericordia University, USA
Thermal therapies have been constitutive components of most ancient systems of medicine and their use is re-emerging. New
evidence has captured the interest in the use of therapeutic heat for its ability to sensitize aberrant cells to radiation injury,
provide costimulatory signals to stir immunocompetence, to precondition tissue in defense against various endogenous injury
and to downregulate pro-inflammatory genes. Copious studies have investigated the modulation of both local and systemic
inflammation by exogenous, local or systemic heat applications and these modalities should reclaim their place in the physical
medicine shack of available therapeutic tools. The induction of heat stress markedly elevates tissue expression of many heat
shock proteins which comprise a superfamily of molecular chaperones found in most tissues. Heat shock proteins are highly
cytoprotective molecules eliciting the appearance of defended tissue phenotypes against several injurious subcellular stresses.
The heat shock response (HSR) can powerfully modulate inflammation by triggering over expression of several heat shock
protein which in turn mediate the inhibiting expression level of factors such as NFkB and thus a cascade of pro-inflammatory
gene profiles. In this presentation, we review the biology of thermal stresses, the current evidence substantiating the uses of
heat as an adjunct therapy in several pathological processes with a focus on inflammation.