Abstract

Traumatic optic neuropathy (TON) is an exceptional vision-threatening clutter that can be caused by visual or head injury and is categorized into coordinate and backhanded TON. The overall incidence of
TON is 0.7–2.5%, and circuitous TON includes a higher predominance than direct TON. Discovery of an afferent pupillary deformity within the nearness of an intaglio globe in an understanding with visual or head injury with decreased visual keenness emphatically recommends TON. Be that as it may, afferent pupillary absconds may be troublesome to distinguish in patients who have gotten narcotics that cause pupillary choking and in those with reciprocal TON. Mechanical shearing of the optic nerve axons and wound rot due to prompt ischemia from harm to the optic nerve microcirculation and apoptosis of neurons may be a plausible component. The proper management of TON is disputable. High-dose corticosteroid treatment and decompression of the optic nerve give no additional benefit over perception alone. Intravenous erythropoietin may be a secure and productive treatment for patients with TON.