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Journal of Radiology
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  • Conference Proceeding   
  • OMICS J Radiol , Vol 10(1)

Link ImmunoTherapy in healing mechanism of “Prolonged medical Starvaticon 42-45 days with very small dosage and weak cytotoxic substances

Ponizovskiy MR*
Head of Laboratory Biochemistry and Toxicology, Kiev, Ukraine
*Corresponding Author: Ponizovskiy MR, Head of Laboratory Biochemistry and Toxicology, Kiev, Ukraine

Received: 09-Jan-2021 / Accepted Date: 21-Jan-2021 / Published Date: 27-Jan-2021

Abstract

Treatment by “Prolonged medical Starvation (during 42-45 days)” causes considerable decrease almost of all depots of an organism exhausting organism’s fat and hydrocarbonic depots leading to competition between cancer tissue and an organism for use of remained decreased depot. Protective forces of the organism become stronger due to support with herbal extracts, delivering vitamins and microelements into organism. Increase of fat metabolism from fat depot leads to augmentation GPX and PHGPX in all cells of an organism which neutralize redundant superoxide [O*] and ROS/H2 O2 /free radicals in G1/S phases cellular cycle of cancer cells cycle suppressing excessive proliferative processes of cancer cells causing elimination irrepressible proliferation and cancer cells depression. Influences Link ImmunoTherapy in “Prolonged medical Starvaticon 42 – 45 days with very small dosage weak cytotoxic substances” on depressed cancer cells promote penetration through cellular walls of cancer cells anticancer antibodies against oncoviral substances for suppression Mitosis-Meiosis phase of cancer cellular cycle where haploid Meiosis phase of viral cellular cycle is deprived. Expression Mitosis cellular cycles of all cells incite T lymphocytes via appearance produced immunoglobulins CTLA-4 and PD-1, and resonance waves of cellular capacitors T memory cells learn and remember waves function of viral substances containing in separated haploid Meiosis phase. T memory cells exert T helper cells, and T helper cells stimulate T killer cells for production antibodies against cancer viral substances which is deprived barriering covalent bonds between Mitosis and Meiosis causing loss viral stem cells function

Thus therapeutic targets of new method cancer treatment using combination immunotherapy with small dosage weak cytotoxic substances prevent recurrence of cancer disease after long anticancer chemotherapy and resistance to anticancer drugs.

Keywords: ImmunoTherapy

Biography

I, Ponizovskiy M., was born at 01.08.1936. At 1966 I have graduated from N.I.Pirogov Vinitsa National Medical University. I worked doctor of therapeutic department in Kiev regional hospital. Then I have graduated from Kiev national Institute of post diploma education and Lvov National Medical University, faculties of laboratory clinical diagnostics, laboratory clinical biochemistry, and laboratory toxicology. At 1969 I worked scientific employee at A.V.Palladin Institute of Biochemistry of Ukraine Academy Sciences in the department “Biochemistry of Growth”. At 1972 - 1990 I worked head of clinical and biochemical laboratory of the First Kiev regional hospital, and I have obtained Degree PhD from 1990. At 1990 - 2002 I worked head of toxicological and biochemical Laboratory of the Kiev regional p/n hospital. Since 2002 I have retired. At 2002 I have left Kiev (Ukraine) for Germany where I have published some scientific works.

Abstract citation: Ponizovskiy M.R., Link ImmunoTherapy in healing mechanism of “Prolonged medical Starvaticon 42 – 45 days with very small dosage and weak cytotoxic substances, Cancer Summit 2020, 3rd Global Expo on Cancer and Oncology Research, July 30, 2020, Page 05

Conference URL: https://cancersummit.alliedacademies.com/

Citation: Ponizovskiy MR (2021) Link ImmunoTherapy in healing mechanism of Prolonged medical Starvaticon 42 – 45 days with very small dosage and weak cytotoxic substances. J Entrepren Organiz Manag 10: e120.

Copyright: © 2021 Ponizovskiy MR. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.

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