ISSN: 2165-7904

Journal of Obesity & Weight Loss Therapy
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  • Mini Review   
  • J Obes Weight Loss Ther 2023, Vol 13(5): 568
  • DOI: 10.4172/2165-7904.1000568

Gynoid Obesity: Understanding the Female-Specific Distribution of Excess Body Fat

Ravi Kiran*
Department of Biotechnology, Kalinga Institute of Industrial Technology, India
*Corresponding Author: Ravi Kiran, Department of Biotechnology, Kalinga Institute of Industrial Technology, India, Email: Ravi_ki@gmail.com

Received: 03-May-2023 / Manuscript No. JOWT-23-99445 / Editor assigned: 05-May-2023 / PreQC No. JOWT-23-99445 (PQ) / Reviewed: 19-May-2023 / QC No. JOWT-23-99445 / Revised: 23-May-2023 / Manuscript No. JOWT-23-99445 (R) / Published Date: 30-May-2023 DOI: 10.4172/2165-7904.1000568

Abstract

Obesity affects men and women the specific impact on women particularly in the form of gynoid obesity is a growing concern. Gynoid obesity refers to excess fat accumulation in the lower body, primarily around the hips, buttocks and thighs. This article aims to explore the causes and consequences of gynoid obesity discuss the challenges faced by individuals affected by this condition and propose potential solutions to address this growing issue.

Keywords

Gynoid obesity; Obesity; Weight loss; Hypertension

Introduction

Gynoid obesity often referred to as "pear-shaped" obesity, is characterized by the disproportionate accumulation of fat in the lower body compared to the upper body. This distribution is believed to be influenced by hormonal factors, particularly estrogen. Estrogen promotes the storage of fat in the lower body, while testosterone, more prevalent in men, promotes fat accumulation in the upper body. The genetic predisposition to gynoid obesity, combined with hormonal and metabolic factors, contributes to its development [1].

Causes and risk factors

Several factors contribute to the development of gynoid obesity. Genetic predisposition plays a significant role, as certain individuals are more susceptible to store fat in the lower body. Hormonal imbalances, such as elevated levels of estrogen and insulin resistance, can also contribute to gynoid obesity. Sedentary lifestyles, poor dietary habits, and an energy imbalance (consuming more calories than expended) are other significant risk factors [2].

Consequences of gynoid obesity

While gynoid obesity may seem less concerning than central or abdominal obesity, it is not without its risks. Excessive fat accumulation in the lower body can still lead to health complications. Gynoid obesity is associated with an increased risk of metabolic disorders, including Type 2 diabetes, cardiovascular diseases, and hypertension. It can also have a significant impact on a person's body image, self-esteem, and mental health, leading to decreased quality of life [3].

Challenges faced by individuals with gynoid obesity

Living with gynoid obesity poses several challenges. Body shaming and societal pressure to conform to unrealistic beauty standards can lead to feelings of shame, guilt, and low self-esteem. Finding suitable clothing options can also be a struggle, as the fashion industry often caters to individuals with different body shapes. Moreover, medical professionals and society, in general, may not fully understand the unique challenges faced by those with gynoid obesity, leading to a lack of appropriate support and resources.

Literature Review

Addressing gynoid obesity requires a multifaceted approach that combines lifestyle changes, medical interventions, and societal support. Encouraging regular physical activity, such as strength training and aerobic exercises, can help reduce body fat and improve overall health.

A balanced diet that focuses on nutrient-dense foods, portion control, and reducing calorie intake is essential. Medical interventions, including hormonal therapy and weight loss medications, may be considered in certain cases. Additionally, promoting body positivity, inclusivity, and challenging societal beauty standards can help create a more supportive environment for individuals with gynoid obesity [4].

Gynoid obesity presents a unique set of challenges for individuals affected by this condition. Understanding its causes, consequences, and the associated psychological and physical impacts is crucial in developing effective strategies for prevention and management. By fostering a supportive and inclusive society, promoting body positivity, and providing appropriate medical interventions, we can work towards addressing gynoid obesity and improving the overall well-being of those affected by this condition. It's time to prioritize a comprehensive and compassionate approach to tackle this growing health concern [5,6].

Fat distribution: Gynoid obesity is characterized by an increased deposition of subcutaneous fat in the lower body. This fat tends to be distributed in the hips, buttocks, and thighs, resulting in a pear-shaped appearance.

Hormonal influence: Hormones play a significant role in the development of gynoid obesity. Estrogen, the primary female sex hormone, promotes fat storage in the lower body. Women typically have higher estrogen levels compared to men, which can contribute to the gynoid fat distribution pattern [7].

Genetic predisposition: Genetic factors also play a role in determining body fat distribution. Some individuals may be genetically predisposed to gynoid obesity, making them more likely to accumulate fat in the lower body [8].

Health implications: While gynoid obesity may have less detrimental health effects compared to android obesity, it is still associated with certain health risks. Excess fat accumulation in the lower body can lead to issues such as joint problems, varicose veins, and cellulite. However, it is generally considered to be less harmful than android obesity, which is associated with a higher risk of cardiovascular diseases, diabetes, and metabolic syndrome [9].

Weight loss challenges: Gynoid obesity can present challenges when it comes to weight loss. The fat stored in the lower body is often more resistant to diet and exercise interventions. It may take longer to notice visible changes in the lower body compared to the abdominal region. However, adopting a healthy lifestyle, including a balanced diet and regular exercise, can still contribute to overall weight loss and improved body composition [10].

It's important to note that body fat distribution is influenced by various factors, including genetics, hormones, and lifestyle. While gynoid obesity may have a distinct fat distribution pattern, it is essential to focus on overall health and well-being rather than targeting specific body parts. Consulting with a healthcare professional or a registered dietitian can provide personalized advice and guidance on managing gynoid obesity [11].

Discussion

This study looked at the relationships between DEXA measurements of total body fat and regional fat patterning and cardio-metabolic risk factors in 82 urban-dwelling children aged 6 to 14 years old. Based on DEXA measurements, an A/G fat ratio may be a straightforward method for determining the distribution of body fat associated with an increased risk of insulin resistance [12-14].

Conclusion

In the female population, a significant correlation was found between the A/G ratio and the US measurement of fat thickness. Additionally, the thickness of the subcutaneous fat in the upper stomach district (S4), lower stomach locale (S5), and instinctive stomach fat in the xiphoidumbilical line (S7) could be utilized to foresee the A/G proportion in a setting where DXA is inaccessible. Notwithstanding, it likewise should be expressed that in guys, the fat thickness estimated utilizing US can't be suggested in assessing the A/G proportion; notwithstanding, this requires further investigations that need to match the member rules and severe assessment conventions.

Acknowledgement

None

Conflict of Interest

None

References

  1. Roubenoff R, Kehayias JJ (1991) The meaning and measurement of lean body mass. Nutr Rev 49: 163-175.
  2. Indexed at, Google Scholar, Crossref

  3. Bey L, Hamilton MT (2003) Suppression of skeletal muscle lipoprotein lipase activity during physical inactivity: a molecular reason to maintain daily low-intensity activity. J Physiol 551: 673-682.
  4. Indexed at, Google Scholar, Crossref

  5. Després JP, Moorjani S, Lupien PJ, Tremblay A, Nadeau A, et al. (1990) Després JP (1993). Regional distribution of body fat, plasma lipoproteins, and cardiovascular disease. Arteriosclerosis 10: 497-511.
  6. Indexed at, Google Scholar, Crossref

  7. Jensen MD, Kanaley JA, Roust LR, O'Brien PC, Braun JS, et al. (1993) Assessment of body composition with use of dual-energy X-ray absorptiometry: evaluation and comparison with other methods. Mayo Clin Proc 68: 867-873.
  8. Indexed at, Google Scholar, Crossref

  9. Cnop M, Havel PJ, Utzschneider KM, Carr DB, Sinha MK, et al. (2003) Relationship of adiponectin to body fat distribution, insulin sensitivity and plasma lipoproteins: evidence for independent roles of age and sex. Diabetologia 46: 459-469.
  10. Indexed at, Google Scholar, Crossref

  11. Flier JS (2004) Obesity wars: molecular progress confronts an expanding epidemic. Cell 116: 337-350.
  12. Indexed at, Google Scholar, Crossref

  13. Manolopoulos KN, Karpe F, Frayn KN (2010) Gluteofemoral body fat as a determinant of metabolic health. Int J Obes (Lond) 34: 949-959.
  14. Indexed at, Google Scholar, Crossref

  15. Tchernof A, Després, JP (2013) Pathophysiology of human visceral obesity: an update. Physiological Reviews 93: 359-404.
  16. Indexed at, Google Scholar, Crossref

  17. Tankó LB, Bagger YZ, Alexandersen P, Larsen PJ, Christiansen C (2003) Peripheral adiposity exhibits an independent dominant antiatherogenic effect in elderly women. Circulation 107: 1626-1631.
  18. Indexed at, Google Scholar, Crossref

  19. Snijder MB, Dekker JM, Visser M, Bouter LM, Stehouwer CD, et al. (2003) Associations of hip and thigh circumferences independent of waist circumference with the incidence of type 2 diabetes: the Hoorn Study. Am J Clin Nutr 77: 1192-1197.
  20. Indexed at, Google Scholar, Crossref

  21. Blaak EE (2001) Gender differences in fat metabolism. Curr Opin Clin Nutr Metab Care 4: 499-502.
  22. Indexed at, Google Scholar, Crossref

  23. Pasquali R, Pelusi C, Genghini S, Cacciari M, Gambineri A (2003) Obesity and reproductive disorders in women. Hum Reprod Update 9: 359-372.
  24. Indexed at, Google Scholar, Crossref

  25. Pouliot MC, Després JP, Nadeau A, Moorjani S, Prud'homme D, et al. (1992) Visceral obesity in men: associations with glucose tolerance, plasma insulin, and lipoprotein levels. Diabetes 41: 826-834.
  26. Indexed at, Google Scholar, Crossref

  27. Kuk JL, Katzmarzyk PT, Nichaman MZ, Church TS (2006) Visceral fat is an independent predictor of all-cause mortality in men. Obesity (Silver Spring) 14: 336-341.
  28. Indexed at, Google Scholar, Crossref

Citation: Kiran R (2023) Gynoid Obesity: Understanding the Female-SpecificDistribution of Excess Body Fat. J Obes Weight Loss Ther 13: 568. DOI: 10.4172/2165-7904.1000568

Copyright: © 2023 Kiran R. This is an open-access article distributed under theterms of the Creative Commons Attribution License, which permits unrestricteduse, distribution, and reproduction in any medium, provided the original author andsource are credited.

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