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In 2014, HCC is worldwide the fifth most common cancer in men and the seventh one in women, and it represents the third most frequent cause of cancer death [1]. Most patients with HCC have elevated serum alpha fetoprotein (AFP) levels. The incidence of HCC is highest in Asia and Africa, where the major risk factors are hepatitis B and C, cirrhosis, and exposure to environmental carcinogens, such as aflatoxin [1]. The pathogenesis of HCC is a multistep process. The molecular pathways involved include p53, β-catenin, NF-κB [2], MAPK [3], ERK [4], JNK [5], WNT/beta - catenin pathway [6] and PI3K/Akt [7], but the detailed molecular mechanisms of HCC are still poorly understood. It is more interesting to find the roles of hormones in HCC molecular mechanisms.
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